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2.2 Dysbiosis-induced metabolic disruption and escalation of inflammatory responses CKD patients manifest profound microbial biodiversity attenuation, accompanied by pathological proliferation of urease-synthesizing bacteria and proteolytic bacterial consortia

Mitochondrial dysfunction contributes to excessive production of mitochondrial ROS and mutations in mtDNA, further exacerbating oxidative stress

Osmoregulation requires brain expression of the renal Na-K-2Cl cotransporter NKCC2

[DOI] [PMC free article] [PubMed] [Google Scholar] Uyeda A

Please note that all compounds A-D increased [ 3 H]-thymidine uptake at least at one concentration value

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